🟢 Safe
Oxalate <25 mg/serving
🟡 Caution
Oxalate 25-99 mg/serving
🔴 Avoid
Oxalate ≥100 mg/serving
🟢

Monk fruit sweetener

🟢 Safe — Low Oxalate
0 mg oxalate / serving
Serving: 1 tsp (4 g) | Per 100 g: 0 mg
✓ Medically Reviewed Content reviewed by OxalateWatch Editorial Team. Based on Harvard T.H. Chan SPH oxalate data (2024) and NKF/AUA dietary guidelines.

📊 Nutrition at a Glance

0 mg
Oxalate / serving
Low Oxalate
Sodium / 100g
Pending
Protein / 100g
Pending
Calcium / 100g
Pending

📏 Oxalate by Serving Size

ServingWeightOxalateVerdict
1 tsp4 g0 mg Safe
2 x 1 tsp8 g0 mg Safe
½ x 1 tsp2 g0 mg Safe

🏷️ Tags

Condiments 🟢 Oxalate Safe Low Oxalate Plant-Based

🟢 See all Condiments rated Safe for kidney stones →

🔗 Related Foods

❓ Frequently Asked Questions

Is Monk fruit sweetener safe for kidney stones?

No red flags here. Just 0 mg per 1 tsp — well below the 25 mg danger line. You can keep Monk fruit sweetener in your regular rotation. Data comes from Harvard (2024).

How much oxalate is in Monk fruit sweetener?

The exact figure: 0 mg of oxalate per 1 tsp of Monk fruit sweetener. Works out to 0 mg/100g. This is the number kidney stone patients actually need. Harvard (2024) verified.

Is Monk fruit sweetener low or high oxalate?

Low. 0 mg per 1 tsp puts Monk fruit sweetener firmly in the low-oxalate category. Safe for regular eating if you're managing kidney stones.

What are the best low-oxalate alternatives to Monk fruit sweetener?

Substitutes exist. The safe alternatives listed below are all low-oxalate foods in the same family as Monk fruit sweetener. Grab one of those and you dodge the oxalate while keeping similar flavors.

How reliable is the data for Monk fruit sweetener?

The oxalate number for Monk fruit sweetener — 0 mg per serving — is from Harvard's 2024 lab analysis. That part is solid. The USDA nutrition data (sodium, protein, calcium) is still being matched — we'll update when it's available.

🔍 Explore More About This Food

📚 Data Sources

Oxalate: Harvard T.H. Chan School of Public Health (2024) via OHF.

📖 Key Scientific References

Taylor EN, Curhan GC. Oxalate intake and the risk for nephrolithiasis. J Am Soc Nephrol. 2007;18(7):2198-2204. PMID: 17538185
Borghi L, Schianchi T, Meschi T, et al. Comparison of two diets for the prevention of recurrent stones in idiopathic hypercalciuria. N Engl J Med. 2002;346(2):77-84. DOI: 10.1056/NEJMoa010369
Fink HA, Akornor JW, Garimella PS, et al. Diet, fluid, or supplements for secondary prevention of nephrolithiasis. Eur Urol. 2009;56(1):72-80. DOI: 10.1016/j.eururo.2009.03.031
Ferraro PM, Taylor EN, Gambaro G, Curhan GC. Dietary and lifestyle factors for primary prevention of nephrolithiasis. BMC Nephrol. 2020;21(1):267. PMID: 32652950
Pearle MS, Goldfarb DS, Assimos DG, et al. Medical management of kidney stones: AUA guideline. J Urol. 2014;192(2):316-324. DOI: 10.1016/j.juro.2014.05.006
Liebman M, Costa G. Effects of calcium and magnesium on urinary oxalate excretion after oxalate loads. J Urol. 2000;163(5):1565-1569. PMID: 10751889
Holmes RP, Assimos DG. The impact of dietary oxalate on kidney stone formation. Urol Res. 2004;32(5):311-316. DOI: 10.1007/s00240-004-0437-3
Lin BB, Lin ME, Huang RH, et al. Dietary treatment and fluid intake for prevention of recurrent calcium stones. PLoS One. 2021;16(4):e0250257. PMID: 33872340
This is dietary reference information, NOT medical advice. Talk to your doctor or a registered dietitian before changing your diet. Oxalate tolerance varies by person, stone type, and medication.